STIM2 Contributes to Enhanced Store‐Operated Ca 2+ Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension-Reference-Cited by-同舟云学术

STIM2 Contributes to Enhanced Store‐Operated Ca 2+ Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension

Published:2011-01 Issue:1 Volume:1 Page:84-94
ISSN:2045-8940
Container-title:Pulmonary Circulation
language:en
Short-container-title:Pulm. circ.

Author:

Song Michael Y.¹,Makino Ayako²³,Yuan Jason X.‐J.¹²³

Affiliation:

1. Biomedical Sciences Graduate ProgramSan DiegoLa JollaCaliforniaUSA

2. Department of MedicineUniversity of CaliforniaSan DiegoLa JollaCaliforniaUSA

3. Department of MedicineUniversity of Illinois at ChicagoChicagoIllinoisUSA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Pulmonary and Respiratory Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.4103/2045-8932.78106

Reference65 articles.

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2. Inflammation, growth factors, and pulmonary vascular remodeling;Hassoun PM;J Am Coll Cardiol,2009

3. Idiopathicpulmonary arterial hypertension;Firth AL;Dis Model Mech,2010

4. Knockdownof stromal interaction molecule 1 attenuates store‐operated Ca2+ entry and Ca2+ responses to acute hypoxia in pulmonary arterial smooth muscle;Lu W;Am J Physiol Lung Cell Mol Physiol,2009

5. Differencesin STIM1 and TRPC expression in proximal and distal pulmonary arterial smooth muscle are associated with differences in Ca2+ responses to hypoxia;Lu W;Am J Physiol Lung Cell Mol Physiol,2008

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