Activation of hexosamine pathway impairs nitric oxide (NO)-dependent arteriolar dilations by increased protein O-GlcNAcylation
Author:
Publisher
Elsevier BV
Subject
Pharmacology,Molecular Medicine,Physiology
Reference38 articles.
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3. Microvascular dysfunction after transient high glucose is caused by superoxide-dependent reduction in the bioavailability of NO and BH(4);Bagi;Am. J. Physiol. Heart Circ. Physiol.,2004
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