Propofol ameliorates endothelial inflammation induced by hypoxia/reoxygenation in human umbilical vein endothelial cells: Role of phosphatase A2-Reference-Cited by-同舟云学术

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Propofol ameliorates endothelial inflammation induced by hypoxia/reoxygenation in human umbilical vein endothelial cells: Role of phosphatase A2

Published:2015-10 Issue: Volume:73 Page:149-157
ISSN:1537-1891
Container-title:Vascular Pharmacology
language:en
Short-container-title:Vascular Pharmacology

Author:

Zhu Minmin,Ding Juan,Jiang Hui,Kong Lingchao,Sun Zhirong,Chen Jiawei,Miao Changhong

Funder

Natural Science Foundation of Shanghai

Publisher

Elsevier BV

Subject

Pharmacology,Molecular Medicine,Physiology

Reference38 articles.

1. Hypoxia-mediated induction of endothelial cell interleukin-1: an autocrine mechanism promoting expression of leukocyte adhesion molecules on the vessel surface;Shreeniwas;J. Clin. Invest.,1992

2. Hypoxia/reoxygenation stimulates endothelial cells to promote interleukin-1 and interleukin-6 production: effects of free radical scavengers;Ala;Agents Actions,1992

3. Nitric oxide attenuates reoxygenation-induced ICAM-1 expression in coronary microvascular endothelium: role of NFkappaB;Kupatt;J. Mol. Cell. Cardiol.,1997

4. Molecular mechanisms of anoxia/reoxygenation-induced neutrophil adherence to cultured endothelial cells;Ichikawa;Circ. Res.,1997

5. Leukocyte adhesion molecules on the vascular endothelium: their role in the pathogenesis of cardiovascular disease and the mechanisms underlying their expression;Sluiter;J. Cardiovasc. Pharmacol.,1993

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