Activation of the KCa3.1 channel contributes to traumatic scratch injury-induced reactive astrogliosis through the JNK/c-Jun signaling pathway-Reference-Cited by-同舟云学术

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Activation of the KCa3.1 channel contributes to traumatic scratch injury-induced reactive astrogliosis through the JNK/c-Jun signaling pathway

Published:2016-06 Issue: Volume:624 Page:62-71
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Yi Mengni,Dou Fangfang,Lu Qin,Yu Zhihua,Chen Hongzhuan

Funder

Science and Technology Commission of Shanghai Municipality

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

General Neuroscience

Reference23 articles.

1. Role of astrocytes in neurovascular coupling;Petzold;Neuron,2011

2. Molecular dissection of reactive astrogliosis and glial scar formation;Sofroniew;Trends Neurosci.,2009

3. Reactive astrocytes in neural repair and protection;Sofroniew;Neuroscientist,2005

4. Selective inhibition of kca3: 1 channels mediates adenosine regulation of the motility of human t cells;Chimote;J. Immunol.,2013

5. Il-4 type 1 receptor signaling up-regulates kcnn4 expression, and increases the kca3.1 current and its contribution to migration of alternative-activated microglia;Ferreira;Front. Cell. Neurosci.,2014

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1. Circular RNA HIPK2 Promotes A1 Astrocyte Activation after Spinal Cord Injury through Autophagy and Endoplasmic Reticulum Stress by Modulating miR-124-3p-Mediated Smad2 Repression;ACS Omega;2023-12-26

2. TRPV1 regulates ApoE4-disrupted intracellular lipid homeostasis and decreases synaptic phagocytosis by microglia;Experimental & Molecular Medicine;2023-02-01

3. TRPV1-Mediated Microglial Autophagy Attenuates Alzheimer’s Disease-Associated Pathology and Cognitive Decline;Frontiers in Pharmacology;2022-01-18

4. KCa3.1 inhibition-induced activation of the JNK/c-Jun signaling pathway enhances IL-10 expression in peripherally-induced regulatory T cells;Journal of Pharmacological Sciences;2022-01

5. β‐Asarone suppresses TNF‐α expression through DNA methylation and c‐Jun‐mediated transcription modulation in scratch‐injured neuronal cells;Journal of Biochemical and Molecular Toxicology;2021-05-09

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