Acute decrease in alkaline phosphatase after brain injury: A potential mechanism for tauopathy-Reference-Cited by-同舟云学术

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Acute decrease in alkaline phosphatase after brain injury: A potential mechanism for tauopathy

Published:2015-11 Issue: Volume:609 Page:152-158
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Arun Peethambaran,Oguntayo Samuel,Albert Stephen Van,Gist Irene,Wang Ying,Nambiar Madhusoodana P.,Long Joseph B.

Funder

Congressionally Directed Medical Research Program

Publisher

Elsevier BV

Subject

General Neuroscience

Reference36 articles.

1. Distinct patterns of expression of traumatic brain injury biomarkers after blast exposure: role of compromised cell membrane integrity;Arun;Neurosci. Lett.,2013

2. Rapid release of tissue enzymes into blood after blast exposure: potential use as biological dosimeters;Arun;PloS one,2012

3. The pattern of human tau phosphorylation is the result of priming and feedback events in primary hippocampal neurons;Bertrand;Neuroscience,2010

4. Modifications in a flexible surface loop modulate the isozyme-specific properties of mammalian alkaline phosphatases;Bossi;J. Biol. Chem.,1993

5. Beta-amyloid fibrils induce tau phosphorylation and loss of microtubule binding;Busciglio;Neuron,1995

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1. МЕХАНІЗМИ РАННЬОГО ГОСТРОГО ПЕРІОДУ ВИБУХОВОЇ ЛЕГКОЇ ЧЕРЕПНО- МОЗКОВОЇ ТРАВМИ;Grail of Science;2024-06-26

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3. The Neurovascular Unit as a Locus of Injury in Low-Level Blast-Induced Neurotrauma;International Journal of Molecular Sciences;2024-01-17

4. Spatiotemporal profile of atrophy in the first year following moderate‐severe traumatic brain injury;Human Brain Mapping;2023-07-03

5. TNAP and P2X7R: New Plasma Biomarkers for Alzheimer’s Disease;International Journal of Molecular Sciences;2023-06-30

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