The expression of NLRX1 in C57BL/6 mice cochlear hair cells: Possible relation to aging- and neomycin-induced deafness
Author:
Funder
National Natural Science Foundation of China
Publisher
Elsevier BV
Subject
General Neuroscience
Reference33 articles.
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2. NLRX1 is a mitochondrial NOD-like receptor that amplifies NF-kappaB and JNK pathways by inducing reactive oxygen species production;Tattoli;EMBO Rep.,2008
3. Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1;Abdul-Sater;J. Biol. Chem.,2010
4. Negative regulation of MAVS-mediated innate immune response by PSMA7;Jia;J. Immunol.,2009
5. Non-Inflammasome forming NLRs in inflammation and tumorigenesis;Allen;Front. Immunol.,2014
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3. NLRX1 knockdown attenuates pro-apoptotic signaling and cell death in pulmonary hyperoxic acute injury;2022-10-28
4. Impacts of impaired mitochondrial dynamics in hearing loss: Potential therapeutic targets;Frontiers in Neuroscience;2022-10-05
5. Mechanism of NLRX1 mediating JNK apoptosis pathway to participate in functional impairment of cochlear hair cells in mice with senile deafness;Cellular and Molecular Biology;2022-09-30
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