S-nitrosylation of GAD65 is implicated in decreased GAD activity and oxygen-induced seizures
Author:
Funder
Office of Naval Research
Publisher
Elsevier BV
Subject
General Neuroscience
Reference32 articles.
1. Enzymatic mechanisms regulating protein S-nitrosylation: implications in health and disease;Anand;J. Mol. Med. (Berl.),2012
2. Mice lacking the 65kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures;Asada;Biochem. Biophys. Res. Commun.,1996
3. Relationship between protein nitration and oxidation and development of hyperoxic seizures;Chavko;Nitric Oxide,2003
4. Changes in brain potentials during convulsions induced by oxygen under pressure;Cohn;J. Neurophysiol.,1945
5. Oxygen-induced seizures and inhibition of human glutamate decarboxylase and porcine cysteine sulfinic acid decarboxylase by oxygen and nitric oxide;Davis;J. Biomed. Sci.,2001
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