Capsaicin-evoked substance P release in rat dorsal horn increases after peripheral inflammation: a microdialysis study
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference32 articles.
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4. Tachykinin multiplicity in rat central nervous system as studied using antisera raised against substance P and neurokinin A;Brodin;Regul. Pept.,1986
5. Adjuvant-induced joint inflammation causes very rapid transcription of beta-preprotachykinin and alpha-CGRP genes in innervating sensory ganglia;Bulling;J. Neurochem.,2001
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1. The Role of Substance P in the Regulation of Bone and Cartilage Metabolic Activity;Frontiers in Endocrinology;2020-02-28
2. Effectiveness of High-Frequency Electrical Stimulation Following Sensitization With Capsaicin;The Journal of Pain;2015-07
3. Substance P Exacerbates Dopaminergic Neurodegeneration through Neurokinin-1 Receptor-Independent Activation of Microglial NADPH Oxidase;Journal of Neuroscience;2014-09-10
4. Inflammation enhances Y1 receptor signaling, neuropeptide Y-mediated inhibition of hyperalgesia, and substance P release from primary afferent neurons;Neuroscience;2014-01
5. Experimental colitis triggers the release of substance P and calcitonin gene-related peptide in the urinary bladder via TRPV1 signaling pathways;Experimental Neurology;2010-10
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