Protection of vincristine-induced neuropathy by WldS expression and the independence of the activity of Nmnat1
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference22 articles.
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2. An 85-kb tandem triplication in the slow Wallerian degeneration (WldS) mouse;Coleman;Proc. Natl. Acad. Sci. U.S.A.,1998
3. Axon pathology in neurological disease: a neglected therapeutic target;Coleman;Trend. Neurosci.,2002
4. NAD(+) and axon degeneration revisited: Nmnat1 cannot substitute for WldS to delay Wallerian degeneration;Conforti;Cell Death Differ.,2006
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2. Study of 300,486 individuals identifies 148 independent genetic loci influencing general cognitive function;Nature Communications;2018-05-29
3. Heparanase attenuates axon degeneration following sciatic nerve transection;Scientific Reports;2018-03-26
4. Targeted deletion of Nmnat1 in mouse retina leads to early severe retinal dystrophy;2017-10-29
5. Impact of Genetic Reduction of NMNAT2 on Chemotherapy-Induced Losses in Cell Viability In Vitro and Peripheral Neuropathy In Vivo;PLOS ONE;2016-01-25
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