Xanthoceraside rescues learning and memory deficits through attenuating beta-amyloid deposition and tau hyperphosphorylation in APP mice-Reference-Cited by-同舟云学术

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Xanthoceraside rescues learning and memory deficits through attenuating beta-amyloid deposition and tau hyperphosphorylation in APP mice

Published:2014-06 Issue: Volume:573 Page:58-63
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Jin Ge,Wang Li-Hua,Ji Xue-Fei,Chi Tian-Yan,Qi Yue,Jiao Qing,Xu Qian,Zhou Xiao-Yu,Zhang Rui,Zou Li-Bo

Funder

National Natural Science Foundation of China

Doctoral Program of Higher Education

National Science and Technology Major Special Project on Major New Drug Innovation of China

Publisher

Elsevier BV

Subject

General Neuroscience

Reference29 articles.

1. Bone marrow-derived mesenchymal stem cells reduce brain amyloid β deposition and accelerate the activation of microglia in an acutely induced Alzheimer's disease mouse model;Lee;Neurosci. Lett.,2009

2. Amyloid-beta, tau and dementia;Takashima;J. Alzheimer's Dis.,2009

3. The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics;Hardy;Science,2002

4. The role of cerebral amyloid beta accumulation in common forms of Alzheimer disease;Gandy;J. Clin. Invest.,2005

5. β-Amyloid precursor protein and tau protein levels are differently regulated in human cerebellum compared to brain regions vulnerable to Alzheimer's type neurodegeneration;Čaušević;Neurosci. Lett.,2010

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4. Xanthoceraside exerts anti-Alzheimer's disease effect by remodeling gut microbiota and modulating microbial-derived metabolites level in rats;Phytomedicine;2022-04

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