Spinal neurochemical mechanisms of acute stress-induced visceral hypersensitivity in healthy rats
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
General Neuroscience
Reference44 articles.
1. Neonatal bladder inflammation alters the role of the central amygdala in hypersensitivity produced by Acute Footshock stress in adult female rats;Ness;Brain Res.,2018
2. Lesions of the central amygdala and ventromedial medulla reduce bladder hypersensitivity produced by acute but not chronic foot shock;Randich;Brain Res.,2017
3. The amygdala central nucleus is required for acute stress-induced bladder hyperalgesia in a rat visceral pain model;DeBerry;Brain Res.,2015
4. Footshock stress differentially affects responses of two subpopulations of spinal dorsal horn neurons to urinary bladder distension in rats;Robbins;Brain Res.,2011
5. Footshock-induced urinary bladder hypersensitivity: role of spinal corticotropin-releasing factor receptors;Robbins;J. Pain,2008
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Pharmacological activation of the amygdala, but not single prolonged footshock-induced acute stress, interferes with cue-induced motivation toward food rewards in rats;Frontiers in Behavioral Neuroscience;2023-09-14
2. Stress-induced bladder hypersensitivity: Effect of corticotropin releasing factor receptors assessed by spinal neurophysiology and neurochemistry;Neuropharmacology;2023-02
3. Neonatal cystitis leads to alterations in spinal corticotropin releasing factor receptor-type 2 content and function in adult rats following bladder re-inflammation;Brain Research;2022-08
4. Neonatal cystitis alters mechanisms of stress-induced visceral hypersensitivity in rats;Neuroscience Letters;2022-05
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