Pulpar tooth injury induces plastic changes in S100B positive astroglial cells in the trigeminal subnucleus caudalis-Reference-Cited by-同舟云学术

Pulpar tooth injury induces plastic changes in S100B positive astroglial cells in the trigeminal subnucleus caudalis

Published:2010-02 Issue:1 Volume:470 Page:71-75
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Canzobre Mariela C.,Ríos Hugo

Publisher

Elsevier BV

Subject

General Neuroscience

Reference36 articles.

1. S100B-stimulated NO production by BV-2 microglia is independent of RAGE transducing activity but dependent on RAGE extracellular domain;Adami;Biochim. Biophys. Acta,2004

2. Could glial activation be a factor in migraine?;Bartley;Med. Hypotheses,2009

3. S100B/RAGE-dependent activation of microglia via NF-kappaB and AP-1 co-regulation of COX-2 expression by S100B, IL-1beta and TNF-alpha;Bianchi;Neurobiol. Aging,2008

4. S100B protein regulates astrocyte shape and migration via interaction with Src kinase;Brozzi;J. Biol. Chem.,2009

5. Dental injury models: experimental tools for understanding neuroinflammatory interactions and polymodal nociceptor functions;Byers;Crit Rev. Oral Biol. Med.,1999

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1. Electroacupuncture exerts prolonged analgesic and neuroprotective effects in a persistent dental pain model induced by multiple dental pulp injuries: GABAergic interneurons-astrocytes interaction;Frontiers in Immunology;2023-10-26

2. S100B single nucleotide polymorphisms exhibit sex-specific associations with chronic pain in sickle cell disease in a largely African-American cohort;PLOS ONE;2020-05-07

3. Occlusion, temporomandibular disorders, and orofacial pain: An evidence-based overview and update with recommendations;The Journal of Prosthetic Dentistry;2018-11

4. Role of Ginkgo biloba extract as an adjunctive treatment of elderly patients with depression and on the expression of serum S100B;Medicine;2018-09

5. Role of Extracellular Damage-Associated Molecular Pattern Molecules (DAMPs) as Mediators of Persistent Pain;Progress in Molecular Biology and Translational Science;2015