Suppression of experimental allergic encephalomyelitis in rats by 50-nT, 7-Hz amplitude-modulated nocturnal magnetic fields depends on when after inoculation the fields are applied-Reference-Cited by-同舟云学术

Suppression of experimental allergic encephalomyelitis in rats by 50-nT, 7-Hz amplitude-modulated nocturnal magnetic fields depends on when after inoculation the fields are applied

Published:2004-11 Issue:2-3 Volume:370 Page:166-170
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Kinoshameg S.A.,Persinger M.A.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference21 articles.

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3. Time course changes of nerve growth factor, corticotrophin-releasing hormone, and nitric oxide synthase isoforms and their possible role in the development of inflammatory response in experimental allergic encephalomyelitis;Calza;Proc. Natl. Acad. Sci. U.S.A.,1997

4. Suppression of experimental allergic encephalomyelitis is specific to frequency and intensity of nocturnally applied, intermittent magnetic fields in rats;Cook;Neurosci. Lett.,2000

5. Infiltration of lymphocytes in the limbic brain following stimulation of subclinical cellular immunity and low dosages of lithium and a cholinergic agent;Cook;Toxicol. Lett.,1999

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1. Extremely low frequency magnetic field induced changes in motor behaviour of gerbils submitted to global cerebral ischemia;Behavioural Brain Research;2012-03

2. Emerging Synergisms Between Drugs and Physiologically-Patterned Weak Magnetic Fields: Implications for Neuropharmacology and the Human Population in the Twenty-First Century;Current Neuropharmacology;2007-12-01

3. Developmental effects of perinatal exposure to extremely weak 7 Hz magnetic fields and nitric oxide modulation in the Wistar albino rat;International Journal of Developmental Neuroscience;2007-09-11

4. A potential multiple resonance mechanism by which weak magnetic fields affect molecules and medical problems: The example of melatonin and experimental “multiple sclerosis”;Medical Hypotheses;2006-01