How mutations shape p53 interactions with the genome to promote tumorigenesis and drug resistance-Reference-Cited by-同舟云学术

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How mutations shape p53 interactions with the genome to promote tumorigenesis and drug resistance

Published:2018-05 Issue: Volume:38 Page:27-43
ISSN:1368-7646
Container-title:Drug Resistance Updates
language:en
Short-container-title:Drug Resistance Updates

Author:

Stiewe Thorsten,Haran Tali E.

Funder

European Research Council (ERC)

Israel Science Foundation

Israel Cancer Association

Publisher

Elsevier BV

Subject

Pharmacology (medical),Infectious Diseases,Cancer Research,Pharmacology,Oncology

Reference245 articles.

1. Mdm2 is required for survival of hematopoietic stem cells/progenitors via dampening of ROS-induced p53 activity;Abbas;Cell. Stem Cell.,2010

2. A Mutant-p53/Smad complex opposes p63 to empower TGF beta-induced metastasis;Adorno;Cell,2009

3. p53 loss-of-heterozygosity is a necessary prerequisite for mutant p53 stabilization and gain-of-function in vivo;Alexandrova;Cell. Death Dis.,2017

4. Depleting stabilized GOF mutant p53 proteins by inhibiting molecular folding chaperones: a new promise in cancer therapy;Alexandrova;Cell. Death Differ.,2017

5. Improving survival by exploiting tumour dependence on stabilized mutant p53 for treatment;Alexandrova;Nature,2015

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