Evaluation of biochemical functions and ventricular performance in regional ischemic-reperfused myocardium by afterload reduction: differential effects of calcium blocking and non-calcium blocking vasodilators-Reference-Cited by-同舟云学术

Evaluation of biochemical functions and ventricular performance in regional ischemic-reperfused myocardium by afterload reduction: differential effects of calcium blocking and non-calcium blocking vasodilators

Published:1988-12 Issue:3 Volume:21 Page:233-246
ISSN:0167-5273
Container-title:International Journal of Cardiology
language:en
Short-container-title:International Journal of Cardiology

Author:

Odom Harrell,Peng Chun-Fu,Murphy Marvin L.,Davis J.Lynn,Straub Karl D.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference41 articles.

1. Intracoronary thrombolysis in acute myocardial infarction: duration of ischemia as a major determinant of late results after recanalization;Schwarz;Am J Cardiol,1982

2. Factors that determine recovery of left ventricular function after thrombolysis in patients with acute myocardial infarction;Sheehan;Circulation,1985

3. Myocardial revascularization after acute infarction;Bolooki;Am J Cardiol,1975

4. Ventricular performance and biochemical alteration of regional ischemic myocardium after reperfusion in the pig;Murphy;Am J Cardiol,1982

5. Contractile and biochemical effects of coronary reperfusion after extended periods of coronary occlusion;Puri;Am J Cardiol,1975

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Ruthenium Red-Catalyzed Degradation of Peroxides Can Prevent Mitochondrial Oxidative Damage Induced by eithertert-Butyl Hydroperoxide or Inorganic Phosphate;Archives of Biochemistry and Biophysics;1998-01

2. Effects of Unloaded Reperfusion on Mitochondrial Function in the Postischemic Myocardium;Artificial Organs;1994-03

3. Do Deficiencies of Endothelial Derived Relaxing Factor Contribute to Myocardial Stunning?;Journal of Cardiac Surgery;1993-03

4. Concanavalin a enhances ATP resynthesis via de novo pathway in postischemic rat hearts;Life Sciences;1993-01