ERK1/2 deactivation enhances cytoplasmic Nur77 expression level and improves the apoptotic effect of fenretinide in human liver cancer cells-Reference-Cited by-同舟云学术

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ERK1/2 deactivation enhances cytoplasmic Nur77 expression level and improves the apoptotic effect of fenretinide in human liver cancer cells

Published:2011-04 Issue:7 Volume:81 Page:910-916
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Yang Hui,Nie Yuqiang,Li Yuyuan,Wan Yu-Jui Yvonne

Funder

NIH

National Natural Science Foundation of China

Bureau of Education of Guangzhou Municipality

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference34 articles.

1. Chemoprevention of experimental carcinogenesis in animals;Moon;Prev Med,1989

2. Mechanisms of fenretinide-induced apoptosis;Hail;Apoptosis,2006

3. Mechanism of fenretinide (4-HPR)-induced cell death;Wu;Apoptosis,2001

4. Angioprevention with fenretinide: targeting angiogenesis in prevention and therapeutic strategies;Sogno;Crit Rev Oncol Hematol,2010

5. Long-term Fenretinide treatment prevents high-fat diet-induced obesity, insulin resistance, and hepatic steatosis;Preitner;Am J Physiol Endocrinol Metab,2009

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2. CDC45 Promotes Proliferation and Glycolysis in Hepatocellular Carcinoma Cells through ERK1/2 Signaling Pathway;J BIOL REG HOMEOS AG;2023

3. Design, synthesis, and evaluation of novel benzoylhydrazone derivatives as Nur77 modulators with potent antitumor activity against hepatocellular carcinoma;Journal of Enzyme Inhibition and Medicinal Chemistry;2023-06-26

4. Discovery of 5-((4-(pyridin-3-yl)pyrimidin-2-yl)amino)-1H-indole-2-carboxamide derivatives as novel anti-cancer agents targeting Nur77;European Journal of Medicinal Chemistry;2022-12

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