Tissue kallikrein is required for the cardioprotective effect of Cyclosporin A in myocardial ischemia in the mouse-Reference-Cited by-同舟云学术

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Tissue kallikrein is required for the cardioprotective effect of Cyclosporin A in myocardial ischemia in the mouse

Published:2015-03 Issue:1 Volume:94 Page:22-29
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Youcef G.,Belaidi E.,Waeckel L.,Fazal L.,Clemessy M.,Vincent M.P.,Zadigue G.,Richer C.,Alhenc-Gelas F.,Ovize M.,Pizard A.

Funder

Inserm

Universities Paris-Descartes and Pierre and Marie Curie

French National Research Agency

European Union-FP 6 framework program

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference45 articles.

1. Myocardial ‘stunning’ in man;Bolli;Circulation,1992

2. Myocardial ischemia: current concepts and future perspectives;Shimokawa;J Cardiol,2008

3. Tissue kallikrein elicits cardioprotection by direct kinin b2 receptor activation independent of kinin formation;Chao;Hypertension,2008

4. Protection by Cyclosporin A of ischemia/reperfusion-induced damage in isolated rat hearts;Griffiths;J Mol Cell Cardiol,1993

5. Role of tissue kallikrein in the cardioprotective effects of ischemic and pharmacological preconditioning in myocardial ischemia;Griol-Charhbili;FASEB J,2005

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1. Imbalance Between Angiotensin II and Kallikrein in Patients With ST-Segment Elevation Myocardial Infarction: A Case-Control Emergency Room Study;Angiology;2024-02-05

2. Preclinical systematic review & meta-analysis of cyclosporine for the treatment of myocardial ischemia-reperfusion injury;Annals of Translational Medicine;2022-09

3. Kallikrein/K1, Kinins, and ACE/Kininase II in Homeostasis and in Disease Insight From Human and Experimental Genetic Studies, Therapeutic Implication;Frontiers in Medicine;2019-06-27

4. Cyclophilin D, Somehow a Master Regulator of Mitochondrial Function;Biomolecules;2018-12-14

5. WDR26/MIP2 interacts with VDAC1 and regulates VDAC1 expression levels in H9c2 cells;Free Radical Biology and Medicine;2018-03

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