MAPKAP kinases MK2 and MK3 in inflammation: Complex regulation of TNF biosynthesis via expression and phosphorylation of tristetraprolin-Reference-Cited by-同舟云学术

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MAPKAP kinases MK2 and MK3 in inflammation: Complex regulation of TNF biosynthesis via expression and phosphorylation of tristetraprolin

Published:2010-12 Issue:12 Volume:80 Page:1915-1920
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Ronkina Natalia,Menon Manoj B.,Schwermann Jessica,Tiedje Christopher,Hitti Edward,Kotlyarov Alexey,Gaestel Matthias

Funder

Deutsche Forschungsgemeinschaft

Hannover Biomedical Research School

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference61 articles.

1. Targeting innate immunity protein kinase signalling in inflammation;Gaestel;Nat Rev Drug Discov,2009

2. Targeting protein kinases for the development of anti-inflammatory drugs;Cohen;Curr Opin Cell Biol,2009

3. Feedback control of the protein kinase TAK1 by SAPK2a/p38alpha;Cheung;EMBO J,2003

4. Role of p38 and JNK mitogen-activated protein kinases in the activation of ternary complex factors;Whitmarsh;Mol Cell Biol,1997

5. p38alpha MAPK inhibits JNK activation and collaborates with IkappaB kinase 2 to prevent endotoxin-induced liver failure;Heinrichsdorff;EMBO Rep,2008

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