β1-Adrenergic receptor-mediated HO-1 induction, via PI3K and p38 MAPK, by isoproterenol in RAW 264.7 cells leads to inhibition of HMGB1 release in LPS-activated RAW 264.7 cells and increases in survival rate of CLP-induced septic mice-Reference-Cited by-同舟云学术

β1-Adrenergic receptor-mediated HO-1 induction, via PI3K and p38 MAPK, by isoproterenol in RAW 264.7 cells leads to inhibition of HMGB1 release in LPS-activated RAW 264.7 cells and increases in survival rate of CLP-induced septic mice

Published:2011-10 Issue:7 Volume:82 Page:769-777
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Ha Yu Mi,Ham Sun Ah,Kim Young Min,Lee Young Soo,Kim Hye Jung,Seo Han Geuk,Lee Jae Heun,Park Min Kyu,Chang Ki Churl

Funder

NRF

Korea Research Foundation

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference42 articles.

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4. Detection of choline-acetyltransferase activity in lymphocytes;Rinner;J Neurosci Res,1993

5. Endogenous norepinephrine regulates tumor necrosis factor-α production from macrophages in vitro;Spengler;J Immunol,1994

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