AMPK upregulates KCa2.3 channels and ameliorates endothelial dysfunction in diet-induced obese mice-Reference-Cited by-同舟云学术

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AMPK upregulates KCa2.3 channels and ameliorates endothelial dysfunction in diet-induced obese mice

Published:2021-01 Issue: Volume:183 Page:114337
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Pang Zheng-Da,Wang Yan,Song Zheng,She Gang,Ma Xiao-Zhen,Sun Xia,Wu Wei,Lai Bao-Chang,Zhang Jiao,Zhang Yi,Du Xiao-Jun,Shyy John Y.J.,Deng Xiu-Ling

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference41 articles.

1. Endothelium-dependent hyperpolarization and endothelial dysfunction;Félétou;J Cardiovasc Pharmacol.,2016

2. Disruption of physiological balance between nitric oxide and endothelium dependent hyperpolarization impairs cardiovascular homeostasis in mice;Godo;Arterioscler Thromb Vasc Biol.,2016

3. Endothelium-dependent hyperpolarization: age, gender and blood pressure, do they matter?;Leung;Acta Physiol.,2017

4. Genetic deficit of SK3 and IK1 channels disrupts the endothelium derived hyperpolarizing factor vasodilator pathway and causes hypertension;Brähler;Circulation,2009

5. Selective blockade of endothelial Ca2+ activated small and intermediate conductance K+ channels suppress EDHF mediated vasodilation;Eichler;Br J Pharmacol.,2003

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3. Endothelial K _Ca 3.1 and K _Ca 2.3 Mediate S1P (Sphingosine-1-Phosphate)–Dependent Vasodilation and Blood Pressure Homeostasis;Arteriosclerosis, Thrombosis, and Vascular Biology;2023-05

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