Overexpression of ABCB1 and ABCG2 contributes to reduced efficacy of the PI3K/mTOR inhibitor samotolisib (LY3023414) in cancer cell lines-Reference-Cited by-同舟云学术

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Overexpression of ABCB1 and ABCG2 contributes to reduced efficacy of the PI3K/mTOR inhibitor samotolisib (LY3023414) in cancer cell lines

Published:2020-10 Issue: Volume:180 Page:114137
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Wu Chung-Pu,Hung Cheng-Yu,Lusvarghi Sabrina,Huang Yang-Hui,Tseng Pin-Jung,Hung Tai-Ho,Yu Jau-Song,Ambudkar Suresh V.

Funder

Ministry of Science and Technology of Taiwan

Chang Gung Medical Research Program

National Institutes of Health

National Cancer Institute

Center for Cancer Research

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference105 articles.

1. PI3K/Akt/mTOR inhibitors in cancer: At the bench and bedside;Alzahrani;Semin. Cancer Biol.,2019

2. Activation of the PI3K/Akt pathway and chemotherapeutic resistance;West;Drug Resist Updat.,2002

3. Roles of the RAF/MEK/ERK and PI3K/PTEN/AKT pathways in malignant transformation and drug resistance;McCubrey;Adv. Enzyme Regul.,2006

4. PTEN/PI3K/Akt pathway regulates the side population phenotype and ABCG2 activity in glioma tumor stem-like cells;Bleau;Cell Stem Cell,2009

5. Overcoming acquired resistance to anticancer therapy: focus on the PI3K/AKT/mTOR pathway;Burris;Cancer Chemother. Pharmacol.,2013

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