HDAC3 inhibitor suppresses endothelial-to-mesenchymal transition via modulating inflammatory response in atherosclerosis-Reference-Cited by-同舟云学术

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HDAC3 inhibitor suppresses endothelial-to-mesenchymal transition via modulating inflammatory response in atherosclerosis

Published:2021-10 Issue: Volume:192 Page:114716
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Chen Lifang,Shang Chenxu,Wang Bo,Wang Guan,Jin Zhen,Yao Feng,Yue Zejun,Bai Liang,Wang Rong,Zhao Sihai,Liu Enqi,Wang Weirong

Funder

National Natural Science Foundation of China

Natural Science Foundation of Shaanxi Province

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference38 articles.

1. Genomewide studies of histone deacetylase function in yeast;Bernstein;Proc Natl Acad Sci U S A,2000

2. Histone deacetylases (HDACs): characterization of the classical HDAC family;de Ruijter;Biochem. J.,2003

3. Molecular evolution of the histone deacetylase family: Functional implications of phylogenetic analysis;Gregoretti;J. Mol. Biol.,2004

4. Deletion of histone deacetylase 3 reveals critical roles in S phase progression and DNA damage control;Bhaskara;Mol. Cell,2008

5. Myeloid type I interferon signaling promotes atherosclerosis by stimulating macrophage recruitment to lesions;Goossens;Cell Metab.,2010

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