Exposure of neuroblastoma cell lines to imatinib results in the upregulation of the CDK inhibitor p27KIP1 as a consequence of c-Abl inhibition-Reference-Cited by-同舟云学术

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Exposure of neuroblastoma cell lines to imatinib results in the upregulation of the CDK inhibitor p27KIP1 as a consequence of c-Abl inhibition

Published:2014-11 Issue:2 Volume:92 Page:235-250
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Lupino Elisa,Ramondetti Cristina,Buccinnà Barbara^ORCID,Piccinini Marco^ORCID

Funder

Progetti di ricerca finanziati dall’Università’ degli Studi di Torino and Regione Piemonte, Ricerca Scientifica Applicata project

TAKTIC consortium

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference50 articles.

1. The Cdk inhibitor p27 in human cancer: prognostic potential and relevance to anticancer therapy;Chu;Nat Rev Cancer,2008

2. The function of p27 KIP1 during tumor development;Lee;Exp Mol Med,2009

3. Deregulated degradation of the cdk inhibitor p27 and malignant transformation;Bloom;Semin Cancer Biol,2003

4. Cdk-inhibitory activity and stability of p27Kip1 are directly regulated by oncogenic tyrosine kinases;Grimmler;Cell,2007

5. Differential modification of p27Kip1 controls its cyclin D-cdk4 inhibitory activity;James;Mol Cell Biol,2008

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3. Second-generation Src/Abl inhibitor bosutinib effectively induces apoptosis in human esophageal squamous cell carcinoma (ESCC) cells via inhibiting Src/Abl signaling;Neoplasma;2020

4. Neuroblastoma Origin and Therapeutic Targets for Immunotherapy;Journal of Immunology Research;2018-07-11

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