Scn3b knockout mice exhibit abnormal ventricular electrophysiological properties-Reference-Cited by-同舟云学术

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Scn3b knockout mice exhibit abnormal ventricular electrophysiological properties

Published:2008-10 Issue:2-3 Volume:98 Page:251-266
ISSN:0079-6107
Container-title:Progress in Biophysics and Molecular Biology
language:en
Short-container-title:Progress in Biophysics and Molecular Biology

Author:

Hakim Parvez,Gurung Iman S.,Pedersen Thomas H.,Thresher Rosemary,Brice Nicola,Lawrence Jason,Grace Andrew A.,Huang Christopher L.-H.

Publisher

Elsevier BV

Subject

Molecular Biology,Biophysics

Reference68 articles.

1. Regulation of the voltage-gated cardiac sodium channel Nav1.5 by interacting proteins;Abriel;Trends Cardiovasc. Med.,2005

2. A novel SCN5A mutation associated with idiopathic ventricular fibrillation without typical ECG findings of Brugada syndrome;Akai;FEBS Lett.,2000

3. The Brugada syndrome: diagnostic criteria and cellular mechanisms;Antzelevitch;Eur Heart J,2001

4. Electrogram prolongation and nifedipine-suppressible ventricular arrhythmias in mice following targeted disruption of KCNE1;Balasubramaniam;J. Physiol.,2003

5. Biophysical phenotypes of SCN5A mutations causing long QT and Brugada syndromes;Baroudi;FEBS Lett.,2000

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