Comment on “The circadian clock gene, BMAL1, promotes radiosensitization in nasopharyngeal carcinoma by inhibiting the epithelial-to-mesenchymal transition via the TGF-β1/Smads/Snail1 axis”-Reference-Cited by-同舟云学术

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Comment on “The circadian clock gene, BMAL1, promotes radiosensitization in nasopharyngeal carcinoma by inhibiting the epithelial-to-mesenchymal transition via the TGF-β1/Smads/Snail1 axis”

Published:2024-08 Issue: Volume:155 Page:106890
ISSN:1368-8375
Container-title:Oral Oncology
language:en
Short-container-title:Oral Oncology

Author:

Prasad Monisha^ORCID,Murugan Ramadurai

Publisher

Elsevier BV

Reference10 articles.

1. The circadian clock gene, BMAL1, promotes radiosensitization in nasopharyngeal carcinoma by inhibiting the epithelial-to-mesenchymal transition via the TGF-β1/Smads/Snail1 axis;Li;Oral Oncol,2024

2. Necdin regulates BMAL1 stability and circadian clock through SGT1-HSP90 chaperone machinery;Lu;Nucleic Acids Res,2020

3. Roles of homologous recombination in response to ionizing radiation-induced DNA damage;Nickoloff;Int J Radiat Biol,2023

4. The positive circadian regulators CLOCK and BMAL1 control G2/M cell cycle transition through Cyclin B1;Farshadi;Cell Cycle,2019

5. Apoptosis as anticancer mechanism: function and dysfunction of its modulators and targeted therapeutic strategies;Pistritto;Aging (Albany NY),2016

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