Mobilization of intracellular calcium by extracellular ATP and by calcium ionophores in the Ehrlich ascites-tumour cell
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Biochemistry,Biophysics
Reference18 articles.
1. Lack of effect of the Ca2+ ionophore A23187 on tumour cells
2. Calcium permeability of Ehrlich ascites tumour cell plasma membrane in vivo
3. Ionomycin acts as an ionophore to release TRH-regulated Ca2+ stores from GH4C1 cells
4. Transport and control of Ca2+ by pigeon erythrocytes. III. A ‘paradoxical’ expulsion of Ca2+ induced by a low dose of A23187 at 0° C
5. Cytosolic Ca2+ homeostasis in Ehrlich and Yoshida carcinomas. A new, membrane-permeant chelator of heavy metals reveals that these ascites tumor cell lines have normal cytosolic free Ca2+.
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2. Effect of Melafen on Structure and Function of Liposome and Ehrlich Ascitic Carcinoma Cell Membranes;Bulletin of Experimental Biology and Medicine;2009-06
3. Mechanical stress induces release of ATP from Ehrlich ascites tumor cells;Biochimica et Biophysica Acta (BBA) - Biomembranes;1999-01
4. Chapter 6 Membrane Events Involved in Volume Regulation;Current Topics in Membranes;1999
5. P2 receptor-mediated signal transduction in Ehrlich ascites tumor cells;Biochimica et Biophysica Acta (BBA) - Biomembranes;1998-09
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