A role for an animal model in determining the immune mechanisms involved in the pathogenesis of rheumatic heart disease
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference11 articles.
1. T cell mimicry in inflammatory heart disease;Cunningham;Mol. Immunol.,2004
2. How an autoimmune reaction triggered by molecular mimicry between streptococcal M protein and cardiac tissue proteins leads to heart lesions in rheumatic heart disease;Fae;J. Autoimm.,2005
3. Molecular analysis of human cardiac myosin-cross-reactive B- and T-cell epitopes of the group A streptococcal M5 protein;Cunningham;Infect. Immun.,1997
4. T-cell reactivity against streptococcal antigens in the periphery mirrors reactivity of heart-infiltrating T lymphocytes in rheumatic heart disease;Guilherme;Infect. Immun.,2001
5. Heart-directed autoimmunity: the case of rheumatic fever;Guilherme;J. Autoimmun.,2001
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2. A biphasic model for full cycle simulation of the human heart aimed at rheumatic heart disease;Computers & Structures;2020-05
3. Group A streptococcal M-protein specific antibodies and T-cells drive the pathology observed in the rat autoimmune valvulitis model;Autoimmunity;2019-02-17
4. An animal model of chronic rheumatic valvulitis induced by formalin-killed streptococci;Rheumatology International;2009-12-12
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