Increased oxidative stress contributes to cardiomyocyte dysfunction and death in patients with Fabry disease cardiomyopathy-Reference-Cited by-同舟云学术

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Increased oxidative stress contributes to cardiomyocyte dysfunction and death in patients with Fabry disease cardiomyopathy

Published:2015-11 Issue:11 Volume:46 Page:1760-1768
ISSN:0046-8177
Container-title:Human Pathology
language:en
Short-container-title:Human Pathology

Author:

Chimenti Cristina,Scopelliti Fernanda,Vulpis Elisabetta,Tafani Marco,Villanova Lidia,Verardo Romina,De Paulis Ruggero,Russo Matteo A.,Frustaci Andrea

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference42 articles.

1. Alpha-galactosidase A deficiency: Fabry disease;Desnick,2001

2. Natural history of Fabry disease in affected males and obligate carrier females;MacDermot;J Inherit Metab Dis,2001

3. Cardiac manifestations of Anderson-Fabry disease in heterozygous females;Kampmann;J Am Coll Cardiol,2002

4. An atypical variant of Fabry's disease in men with left ventricular hypertrophy;Nakao;N Engl J Med,1995

5. Prevalence of Anderson-Fabry disease in male patients with late-onset hypertrophic cardiomyopathy;Sachdev;Circulation,2002

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1. Inflammation, Oxidative Stress, and Endothelial Dysfunction in the Pathogenesis of Vascular Damage: Unraveling Novel Cardiovascular Risk Factors in Fabry Disease;International Journal of Molecular Sciences;2024-07-29

2. Challenging the Conventional Treatment Initiation Paradigm: Early Detection of Irreversible Cellular Damage in Cardiac Biopsies of Fabry Disease Before the Formation of Gb3 Inclusion Bodies;2024-07-01

3. Oxidative stress and its role in Fabry disease;Journal of Nephrology;2024-06-15

4. Inflammation in Fabry disease: stages, molecular pathways, and therapeutic implications;Frontiers in Cardiovascular Medicine;2024-06-12

5. Long‐Term Clinical‐Pathologic Results of Enzyme Replacement Therapy in Prehypertrophic Fabry Disease Cardiomyopathy;Journal of the American Heart Association;2024-04-16

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