A full range of mouse sinoatrial node AP firing rates requires protein kinase A-dependent calcium signaling-Reference-Cited by-同舟云学术

A full range of mouse sinoatrial node AP firing rates requires protein kinase A-dependent calcium signaling

Published:2011-11 Issue:5 Volume:51 Page:730-739
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Liu Jie,Sirenko Syevda,Juhaszova Magdalena,Ziman Bruce,Shetty Veena,Rain Silvia,Shukla Shweta,Spurgeon Harold A.,Vinogradova Tatiana M.,Maltsev Victor A.,Lakatta Edward G.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

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3. Constitutive phosphodiesterase activity restricts spontaneous beating rate of cardiac pacemaker cells by suppressing local Ca2+ releases;Vinogradova;Circ Res,2008

4. Mouse models for studying pacemaker channel function and sinus node arrhythmia;Ludwig;Prog Biophys Mol Biol,2010

5. Sinus node dysfunction following targeted disruption of the murine cardiac sodium channel gene Scn5a;Lei;J Physiol,2005

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