CaMKII and reactive oxygen species contribute to early reperfusion arrhythmias, but oxidation of CaMKIIδ at methionines 281/282 is not a determining factor-Reference-Cited by-同舟云学术

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CaMKII and reactive oxygen species contribute to early reperfusion arrhythmias, but oxidation of CaMKIIδ at methionines 281/282 is not a determining factor

Published:2023-02 Issue: Volume:175 Page:49-61
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Haugsten Hansen Marie,Sadredini Mani,Hasic Almira,Anderson Mark E.,Sjaastad Ivar,Korseberg Stokke Mathis

Funder

Helse Sør-Øst RHF

Norges Idrettshøgskole

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference51 articles.

1. CaMKII-dependent responses to ischemia and reperfusion challenges in the heart;Bell;Front. Pharmacol.,2014

2. Sudden cardiac death: epidemiology and risk factors;Adabag;Nat. Rev. Cardiol.,2010

3. The first minutes of reperfusion: a window of opportunity for cardioprotection;Piper;Cardiovasc. Res.,2004

4. Current mechanistic concepts in ischemia and reperfusion injury;Wu;Cell. Physiol. Biochem.,2018

5. Multitarget strategies to reduce myocardial ischemia/reperfusion injury;Davidson;J. Am. Coll. Cardiol.,2019

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Myocardial oxidative stress is increased in early reperfusion, but systemic antioxidative therapy does not prevent ischemia-reperfusion arrhythmias in pigs;Frontiers in Cardiovascular Medicine;2023-09-26

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