A590T mutation in KCNQ1 C-terminal helix D decreases IKs channel trafficking and function but not Yotiao interaction-Reference-Cited by-同舟云学术

A590T mutation in KCNQ1 C-terminal helix D decreases IKs channel trafficking and function but not Yotiao interaction

Published:2014-07 Issue: Volume:72 Page:273-280
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Kinoshita Koshi,Komatsu Takuto,Nishide Kohki,Hata Yukiko,Hisajima Nozomi,Takahashi Hiroyuki,Kimoto Katsuya,Aonuma Kei,Tsushima Eikichi,Tabata Toshihide,Yoshida Tomoyuki,Mori Hisashi,Nishida Kunihiro,Yamaguchi Yoshiaki,Ichida Fukiko,Fukurotani Kenkichi,Inoue Hiroshi,Nishida Naoki

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

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3. The KCNQ1 potassium channel: from gene to physiological function;Jespersen;Physiology,2005

4. Role of potassium currents in cardiac arrhythmias;Ravens;Europace,2008

5. The long QT syndromes: genetic basis and clinical implications;Chiang;J Am Coll Cardiol,2000

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