Cardiomyocyte-specific expression of CRNK, the C-terminal domain of PYK2, maintains ventricular function and slows ventricular remodeling in a mouse model of dilated cardiomyopathy-Reference-Cited by-同舟云学术

Cardiomyocyte-specific expression of CRNK, the C-terminal domain of PYK2, maintains ventricular function and slows ventricular remodeling in a mouse model of dilated cardiomyopathy

Published:2014-07 Issue: Volume:72 Page:281-291
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Koshman Yevgeniya E.,Chu Miensheng^ORCID,Kim Taehoon,Kalmanson Olivia^ORCID,Farjah Mariam,Kumar Mohit,Lewis William,Geenen David L.,de Tombe Pieter,Goldspink Paul H.,Solaro R. John,Samarel Allen M.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference43 articles.

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3. Pyk2 expression and phosphorylation in neonatal and adult cardiomyocytes;Bayer;J Mol Cell Cardiol,2001

4. Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes;Menashi;Cell Tissue Res,2009

5. Protein kinase Cε-dependent activation of proline-rich tyrosine kinase 2 in neonatal rat ventricular myocytes;Bayer;J Mol Cell Cardiol,2003

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