Blocking endothelial TRPV4-Nox2 interaction helps reduce ROS production and inflammation, and improves vascular function in obese mice-Reference-Cited by-同舟云学术

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Blocking endothelial TRPV4-Nox2 interaction helps reduce ROS production and inflammation, and improves vascular function in obese mice

Published:2021-08 Issue: Volume:157 Page:66-76
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Gao Mengru,Han Jing,Zhu Yifei,Tang Chunlei,Liu Liangliang,Xiao Wang,Ma Xin

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference59 articles.

1. Inflammation, oxidative stress, and obesity;Fernandez-Sanchez;Int. J. Mol. Sci.,2011

2. Relationship between inflammation, the gut microbiota, and metabolic osteoarthritis development: studies in a rat model;Collins;Osteoarthr. Cartil.,2015

3. Nox4 NADPH oxidase mediates peroxynitrite-dependent uncoupling of endothelial nitric-oxide synthase and fibronectin expression in response to angiotensin II: role of mitochondrial reactive oxygen species;Lee;J. Biol. Chem.,2013

4. Intake of omega-3 formulation EPA:DHA 6:1 by old rats for 2 weeks improved endothelium-dependent relaxations and normalized the expression level of ACE/AT1R/NADPH oxidase and the formation of ROS in the mesenteric artery;Farooq;Biochem. Pharmacol.,2020

5. Activation of TRPV4 channels (hVRL-2/mTRP12) by phorbol derivatives;Watanabe;J. Biol. Chem.,2002

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