Class I HDACs control a JIP1-dependent pathway for kinesin-microtubule binding in cardiomyocytes-Reference-Cited by-同舟云学术

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Class I HDACs control a JIP1-dependent pathway for kinesin-microtubule binding in cardiomyocytes

Published:2017-11 Issue: Volume:112 Page:74-82
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Blakeslee Weston W.,Lin Ying-Hsi,Stratton Matthew S.,Tatman Philip D.,Hu Tianjing,Ferguson Bradley S.,McKinsey Timothy A.

Funder

National Institutes of Health

American Heart Association

University of Colorado Denver

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference42 articles.

1. Lysine acetylation targets protein complexes and co-regulates major cellular functions;Choudhary;Science,2009

2. The growing landscape of lysine acetylation links metabolism and cell signalling;Choudhary;Nat Rev Mol Cell Biol,2014

3. Proteomic analysis of lysine acetylation sites in rat tissues reveals organ specificity and subcellular patterns;Lundby;Cell Rep.,2012

4. Molecular evolution of the histone deacetylase family: functional implications of phylogenetic analysis;Gregoretti;J. Mol. Biol.,2004

5. Small-molecule therapies for cardiac hypertrophy: moving beneath the cell surface;McKinsey;Nat. Rev. Drug Discov.,2007

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