Progressive loss of creatine maintains a near normal ΔG∼ATP in transgenic mouse hearts with cardiomyopathy caused by overexpressing Gsα-Reference-Cited by-同舟云学术

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Progressive loss of creatine maintains a near normal ΔG∼ATP in transgenic mouse hearts with cardiomyopathy caused by overexpressing Gsα

Published:2010-04 Issue:4 Volume:48 Page:591-599
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Shen Weiqun,Vatner Dorothy E.,Vatner Stephen F.,Ingwall Joanne S.

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference35 articles.

1. Human myocardial ATP content and in vivo contractile function;Starling;Mol. Cell. Biochem.,1998

2. Progressive loss of myocardial ATP due to a loss of total purine during the development of heart failure in dogs;Shen;Circulation,1999

3. The failing heart—an engine out of fuel;Neubauer;N. Engl. J. Med.,2007

4. Energy metabolism in heart failure and remodeling;Ingwall;Cardiovasc. Res.,2009

5. Energetic basis for reduced contractile reserve in isolated rat hearts;Tian;Am. J. Physiol.,1996

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