Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca2+ currents and promote proarrhythmic behavior in ventricular myocytes-Reference-Cited by-同舟云学术

Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca2+ currents and promote proarrhythmic behavior in ventricular myocytes

Published:2014-09 Issue: Volume:74 Page:115-124
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Limpitikul Worawan B.,Dick Ivy E.,Joshi-Mukherjee Rosy^ORCID,Overgaard Michael T.,George Alfred L.,Yue David T.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference57 articles.

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