Stimulation of ICa by basal PKA activity is facilitated by caveolin-3 in cardiac ventricular myocytes-Reference-Cited by-同舟云学术

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Stimulation of ICa by basal PKA activity is facilitated by caveolin-3 in cardiac ventricular myocytes

Published:2014-03 Issue: Volume:68 Page:47-55
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Bryant Simon,Kimura Tomomi E.,Kong Cherrie H.T.,Watson Judy J.,Chase Anabelle,Suleiman M. Saadeh,James Andrew F.,Orchard Clive H.

Funder

British Heart Foundation

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference27 articles.

1. Mechanism of release of calcium from sarcoplasmic reticulum of guinea-pig cardiac cells;Beuckelmann;J Physiol Lond,1988

2. Simulated calcium current can both cause calcium loading and trigger calcium release from the sarcoplasmic reticulum of a skinned canine cardiac Purkinje cell;Fabiato;J Gen Physiol,1985

3. Excitation–contraction coupling in rat ventricular myocytes after formamide-induced detubulation;Kawai;Am J Physiol,1999

4. Localised Ca channel phosphorylation modulates the distribution of L-type Ca current in cardiac myocytes;Chase;J Mol Cell Cardiol,2010

5. Calcium leak through ryanodine receptors leads to atrial fibrillation in 3 mouse models of catecholaminergic polymorphic ventricular tachycardia;Shan;Circ Res,2012

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3. Role of the T-Tubules in the Control of Cellular Calcium and Inotropic Interventions in Cardiac Ventricular Myocytes;Heart Rate and Rhythm;2023

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