Contribution of the neuronal sodium channel NaV1.8 to sodium- and calcium-dependent cellular proarrhythmia-Reference-Cited by-同舟云学术

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Contribution of the neuronal sodium channel NaV1.8 to sodium- and calcium-dependent cellular proarrhythmia

Published:2020-07 Issue: Volume:144 Page:35-46
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Bengel Philipp,Ahmad Shakil,Tirilomis Petros,Trum Maximilian,Dybkova Nataliya,Wagner Stefan,Maier Lars S.,Hasenfuß Gerd,Sossalla Samuel

Funder

University Hospital Regensburg

Marga und Walter Boll-Stiftung

the Deutsche Forschungsgemeinschaft

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference59 articles.

1. Na channels that remain open throughout the cardiac action potential plateau;Liu;Biophys. J.,1992

2. Novel, ultraslow inactivating sodium current in human ventricular cardiomyocytes;Maltsev;Circulation.,1998

3. Role of late sodium current as a potential arrhythmogenic mechanism in the progression of pressure-induced heart disease;Toischer;J. Mol. Cell. Cardiol.,2013

4. Blocking late sodium current reduces hydrogen peroxide-induced arrhythmogenic activity and contractile dysfunction;Song;J. Pharmacol. Exp. Ther.,2006

5. Altered Na(+) currents in atrial fibrillation effects of ranolazine on arrhythmias and contractility in human atrial myocardium;Sossalla;J. Am. Coll. Cardiol.,2010

Cited by 8 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. NaV1.8 as Proarrhythmic Target in a Ventricular Cardiac Stem Cell Model;International Journal of Molecular Sciences;2024-06-02

2. Molecular and Functional Relevance of NaV1.8-Induced Atrial Arrhythmogenic Triggers in a Human SCN10A Knock-Out Stem Cell Model;International Journal of Molecular Sciences;2023-06-15

3. Functional evaluation of the tachycardia patient‐derived iPSC cardiomyocytes carrying a novel pathogenic SCN5A variant;Journal of Cellular Physiology;2022-08-12

4. Late Sodium Current of the Heart: Where Do We Stand and Where Are We Going?;Pharmaceuticals;2022-02-15

5. Detrimental proarrhythmogenic interaction of Ca2+/calmodulin-dependent protein kinase II and NaV1.8 in heart failure;Nature Communications;2021-11-15

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