p38 MAPK inhibition: A promising therapeutic approach for COVID-19

Author:

Grimes Joseph M.,Grimes Kevin V.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference33 articles.

1. SARS-CoV-2 cell entry depends on ACE2 and TMPRSS2 and Is blocked by a clinically proven protease inhibitor;Hoffmann;Cell,2020

2. Tissue distribution of ACE2 protein, the functional receptor for SARS coronavirus. A first step in understanding SARS pathogenesis;Hamming;J. Pathol.,2004

3. Clinical predictors of mortality due to COVID-19 based on an analysis of data of 150 patients from Wuhan;Ruan;China. Intensive Care Med.,2020

4. Modulation of mitogen-activated protein kinase attenuates sepsis-induced acute lung injury in acute respiratory distress syndrome rats;Fang;Mol. Med. Rep.,2017

5. Inhibition of p38 mitogen-activated protein kinase decreases cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion. circulation;Ma;Am. Heart Assoc.,1999

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