Cardiac-specific deficiency of the mitochondrial calcium uniporter augments fatty acid oxidation and functional reserve-Reference-Cited by-同舟云学术

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Cardiac-specific deficiency of the mitochondrial calcium uniporter augments fatty acid oxidation and functional reserve

Published:2019-02 Issue: Volume:127 Page:223-231
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Altamimi Tariq R.,Karwi Qutuba G.,Uddin Golam Mezbah,Fukushima Arata,Kwong Jennifer Q.,Molkentin Jeffery D.,Lopaschuk Gary D.

Funder

Canadian Institutes of Health Research

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference41 articles.

1. Calcium uptake by rat kidney mitochondria;DeLuca;Proc. Natl. Acad. Sci.,1961

2. The mitochondrial calcium uniporter (MCU): molecular identity and physiological roles;Patron;J. Biol. Chem.,2013

3. A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter;De Stefani;Nature,2011

4. Integrative genomics identifies MCU as an essential component of the mitochondrial calcium uniporter;Baughman;Nature,2011

5. MICU1 encodes a mitochondrial EF hand protein required for Ca(2+) uptake;Perocchi;Nature,2010

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2. Unravelling the complexity of the mitochondrial Ca2+ uniporter: regulation, tissue specificity, and physiological implications;Cell Calcium;2024-07

3. MCU genetically altered mice suggest how mitochondrial Ca2+ regulates metabolism;Trends in Endocrinology & Metabolism;2024-04

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