Overexpression of inducible nitric oxide synthase in the diabetic heart compromises ischemic postconditioning

Author:

Wang Tian-Tian,Shi Mao-Mao,Liao Xiao-Long,Li Yu-Quan,Yuan Hao-Xiang,Li Yan,Liu Xiang,Ning Da-Sheng,Peng Yue-Ming,Yang Fan,Mo Zhi-Wei,Jiang Yu-Mei,Xu Ying-Qi,Li Haobo,Wang Min,Ou Zhi-Jun,Xia Zhengyuan,Ou Jing-Song

Funder

National Natural Science Foundation of China

International Cooperation

Ministry of Science and Technology of China

Guangdong Natural Science Fund Committee

Changjiang Scholars Program from the Ministry of Education of China

Guangdong Pearl River Scholars Program

Sun Yat-sen University Clinical Research 5010 Program

Hong Kong Research Grant Council (RGC)/GRF

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference59 articles.

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2. A comparative study of in-hospital outcome of patients with ST-segment elevation myocardial infarction with and without diabetes mellitus, after thrombolytic therapy; In Government Hospital of Rajkot, Gujarat, India;Hathi;J. Assoc. Physicians India,2017

3. Impact of type 2 diabetes mellitus on recurrent myocardial infarction in China;Li;Diab. Vasc. Dis. Res.,2016

4. Cardioprotective effect of Notch signaling on the development of myocardial infarction complicated by diabetes mellitus;Wu;Exp. Ther. Med.,2017

5. Absence of inducible nitric oxide synthase reduces myocardial damage during ischemia reperfusion in streptozotocin-induced hyperglycemic mice;Marfella;Diabetes,2004

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