Another calcium paradox in heart failure
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine,Molecular Biology
Reference37 articles.
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2. Calcium and cardiomyopathies;Kranias;Subcell Biochem,2007
3. Increased SR Ca2+ cycling contributes to improved contractile performance in SERCA2a-overexpressing transgenic rats;Maier;Cardiovasc Res,2005
4. Intracellular Na(+) concentration is elevated in heart failure but Na/K pump function is unchanged;Despa;Circulation,2002
5. Molecular correlates of altered expression of potassium currents in failing rabbit myocardium;Rose;Am J Physiol Heart Circ Physiol,2005
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1. Cardiac Muscle Membrane Stabilization in Myocardial Reperfusion Injury;JACC: Basic to Translational Science;2019-04
2. The calcium paradox - What should we have to fear?;Revista Brasileira de Cirurgia Cardiovascular;2014
3. SERCA2a gene therapy in heart failure: an anti-arrhythmic positive inotrope;British Journal of Pharmacology;2013-12-10
4. Diastolic transient inward current in long QT syndrome type 3 is caused by Ca2+ overload and inhibited by ranolazine;Journal of Molecular and Cellular Cardiology;2009-08
5. The cardiac IP3 receptor: Uncovering the role of “the other” calcium-release channel;Journal of Molecular and Cellular Cardiology;2008-08
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