The structural basis of alpha-tropomyosin linked (Asp230Asn) familial dilated cardiomyopathy-Reference-Cited by-同舟云学术

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The structural basis of alpha-tropomyosin linked (Asp230Asn) familial dilated cardiomyopathy

Published:2017-07 Issue: Volume:108 Page:127-137
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Lynn M.L.,Tal Grinspan L.,Holeman T.A.,Jimenez J.,Strom J.,Tardiff J.C.

Funder

American Heart Association

Heart, Lung, and Blood Training

NIH

Steven M. Gootter Foundation

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference54 articles.

1. A molecular basis for familial hypertrophic cardiomyopathy: a beta cardiac myosin heavy chain gene missense mutation;Geisterfer-Lowrance;Cell,1990

2. Research priorities in hypertrophic cardiomyopathy: report of a working group of the national heart, lung, and blood institute;Force;Circulation,2010

3. Familial dilated cardiomyopathy caused by an alpha-tropomyosin mutation: the distinctive natural history of sarcomeric dilated cardiomyopathy;Lakdawala;J. Am. Coll. Cardiol.,2010

4. Atomic resolution probe for allostery in the regulatory thin filament;Williams;Proc. Natl. Acad. Sci. U. S. A.,2016

5. Dilated and hypertrophic cardiomyopathy mutations in troponin and α-tropomyosin have opposing effects on the calcium affinity of cardiac thin filaments;Robinson;Circ. Res.,2007

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