ROS generated during early reperfusion contribute to intermittent hypobaric hypoxia-afforded cardioprotection against postischemia-induced Ca2+ overload and contractile dysfunction via the JAK2/STAT3 pathway-Reference-Cited by-同舟云学术

ROS generated during early reperfusion contribute to intermittent hypobaric hypoxia-afforded cardioprotection against postischemia-induced Ca2+ overload and contractile dysfunction via the JAK2/STAT3 pathway

Published:2015-04 Issue: Volume:81 Page:150-161
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Wu Lan,Tan Ji-Liang,Wang Zhi-Hua,Chen Yi-Xiong,Gao Ling,Liu Jin-Long,Shi Yun-Hua,Endoh Masao,Yang Huang-Tian

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

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4. Intermittent hypobaric hypoxia improves post-ischemic recovery of myocardial contractile function via redox signaling during early reperfusion;Wang;Am J Physiol Heart Circ Physiol,2011

5. Concentration-dependent wrestling between detrimental and protective effects of H2O2 during myocardial ischemia/reperfusion;Wang;Cell Death Dis,2014

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