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Ca2+/calmodulin-dependent protein kinase II is essential in hyperacute pressure overload

  • Published:2020-01 Issue: Volume:138 Page:212-221
  • ISSN:0022-2828
  • Container-title:Journal of Molecular and Cellular Cardiology
  • language:en
  • Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Baier Maria J.,Klatt Susanne,Hammer Karin P.,Maier Lars S.,Rokita Adam G.

Funder

Regensburg University ReForM A

DFG

Deutsche Forschungsgemeinschaft

German Cardiac Society

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference54 articles.

1. Heart Disease and Stroke Statistics’2017 Update: A Report from the American Heart Association;Benjamin,2017

2. National Hospital Ambulatory Medical Care Survey: 2016 emergency department summary tables;Rui,2016

3. Transgenic CaMKIIδc overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release;Maier;Circ. Res.,2003

4. Ca2+/calmodulin-dependent protein kinase II regulates cardiac Na+ channels;Wagner;J. Clin. Invest.,2006

5. Calcium/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure;Sag;Circ. Hear. Fail.,2009
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