A biomimetic ion channel shortens the QT interval of type 2 long QT syndrome through efficient transmembrane transport of potassium ions-Reference-Cited by-同舟云学术

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A biomimetic ion channel shortens the QT interval of type 2 long QT syndrome through efficient transmembrane transport of potassium ions

Published:2024-06 Issue: Volume:181 Page:391-401
ISSN:1742-7061
Container-title:Acta Biomaterialia
language:en
Short-container-title:Acta Biomaterialia

Author:

Sun Shuang,Xu Zhaocheng,Lin Ze,Chen Weiwei,Zhang Yue,Yan Mengjie,Ren Shengnan,Liu Qihui,Zhu Huimin,Tian Bin,Zhang Jian,Zhang Weijia,Jiang Shan,Sheng Chuqiao,Ge Jingyan,Chen Fangfang,Dong Zeyuan

Funder

National Key Research and Development Program of China Stem Cell and Translational Research

National Natural Science Foundation of China

China Postdoctoral Science Foundation

Publisher

Elsevier BV

Reference40 articles.

1. Molecular physiology of cardiac repolarization;Nerbonne;Physiol. Rev.,2005

2. Glucose, sulfonylureas, and neurotransmitter release: role of ATP-sensitive K+ channels;Amoroso;Science,1990

3. Y-26763: ATP-sensitive K+ channel activation and the inhibition of insulin release from human pancreatic beta-cells;Cosgrove;Eur. J. Pharmacol.,2004

4. Modulation of ion conductance and active transport by TGF-beta 1 in alveolar epithelial cell monolayers;Willis;Am. J. Physiol.,2003

5. Inhibition of G protein-activated inwardly rectifying K+ channels by Ifenprodil;Kobayashi;Neuropsychopharmacology,2005

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