Human Δ4-3-oxosteroid 5β-reductase (AKR1D1) deficiency and steroid metabolism-Reference-Cited by-同舟云学术

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Human Δ4-3-oxosteroid 5β-reductase (AKR1D1) deficiency and steroid metabolism

Published:2008-04 Issue:4 Volume:73 Page:417-423
ISSN:0039-128X
Container-title:Steroids
language:en
Short-container-title:Steroids

Author:

Palermo Mario,Marazzi Maria Grazia,Hughes Beverly A.,Stewart Paul M.,Clayton Peter T.,Shackleton Cedric H.L.

Publisher

Elsevier BV

Subject

Organic Chemistry,Clinical Biochemistry,Pharmacology,Endocrinology,Molecular Biology,Biochemistry

Reference19 articles.

1. Cloning and expression of cDNA of human Δ4-3-oxosteroid 5β-reductase and substrate specificity of the expressed enzyme;Kondo;Eur J Biochem,1994

2. 3-Oxo-Δ4 bile acids in liver disease;Clayton;Lancet,1988

3. Δ4-3-Oxosteroid 5β-reductase deficiency: failure of ursodeoxycholic acid treatment and response to chenodeoxycholic acid plus cholic acid;Clayton;Gut,1996

4. Δ4-3-Oxosteroid 5β-reductase deficiency described in identical twins with neonatal hepatitis A new inborn error in bile acid synthesis;Setchell;J Clin Investig,1988

5. Bile acid biosynthesis;Russell;Biochemistry,1992

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2. Healthy Patients With AKR1D1 Mutation Not Requiring Primary Bile Acid Therapy: A Case Series;JPGN Reports;2023-10-09

3. Assessment of the inhibitory potential of anabolic steroids towards human AKR1D1 by computational methods and in vitro evaluation;Toxicology Letters;2023-08

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