ER-α36, a novel variant of ERα, is involved in the regulation of Tamoxifen-sensitivity of glioblastoma cells

Author:

Liu Yang,Huang Liang,Guan Xin,Li Hongyan,Zhang Qi-Qi,Han Chao,Wang Ya-Jun,Wang Cui,Zhang Yejun,Qu Chao,Liu Jing,Zou Wei

Funder

National Natural Science Foundation of China

International S&T Cooperation Project of the Ministry of S&T of China

Science and Technology Plan Projects in Laoning Province

Publisher

Elsevier BV

Subject

Organic Chemistry,Clinical Biochemistry,Pharmacology,Endocrinology,Molecular Biology,Biochemistry

Reference29 articles.

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2. Agents with selective estrogen receptor (ER) modulator activity induce apoptosis in vitro and in vivo in ER-negative glioma cells;Hui;Cancer Res.,2004

3. Current chemotherapy for glioblastoma;Parney;Cancer J.,2003

4. Tamoxifen-resistant glioma-cell sub-populations are characterized by increased migration and proliferation;Puchner;Int. J. Cancer,2000

5. Tamoxifen-induced apoptosis of rat C6 glioma cells via PI3K/Akt, JNK and ERK activation;Feng;Oncol. Rep.,2010

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