Depressive, anxiety and hypomanic symptoms in schizophrenia may be driven by tryptophan catabolite (TRYCAT) patterning of IgA and IgM responses directed to TRYCATs-Reference-Cited by-同舟云学术

Depressive, anxiety and hypomanic symptoms in schizophrenia may be driven by tryptophan catabolite (TRYCAT) patterning of IgA and IgM responses directed to TRYCATs

Published:2018-01 Issue: Volume:80 Page:205-216
ISSN:0278-5846
Container-title:Progress in Neuro-Psychopharmacology and Biological Psychiatry
language:en
Short-container-title:Progress in Neuro-Psychopharmacology and Biological Psychiatry

Author:

Kanchanatawan Buranee,Sirivichayakul Sunee^ORCID,Carvalho André F.,Anderson George,Galecki Piotr,Maes Michael

Funder

Asahi Glass Foundation

Chulalongkorn University

Publisher

Elsevier BV

Subject

Biological Psychiatry,Pharmacology

Reference79 articles.

1. Neuronal-immune interactions in mediating stress effects in the etiology and course of schizophrenia: role of the amygdala in developmental co-ordination;Anderson;Med. Hypotheses,2011

2. How immune-inflammatory processes link CNS and psychiatric disorders: classification and treatment implications;Anderson;CNS Neurol. Disord. Drug Targets,2016

3. The interactions of tryptophan and its catabolites with melatonin and the alpha 7 nicotinic receptor in CNS and psychiatric disorders: role for the aryl hydrocarbon receptor and direct mitochondria regulation;Anderson;Int. J. Tryptophan Res.,2017

4. Schizophrenia is primed for an increased expression of depression through activation of immuno-inflammatory, oxidative and nitrosative stress, and tryptophan catabolite pathways;Anderson;Prog. Neuro-Psychopharmacol. Biol. Psychiatry,2013

5. Biological phenotypes underpin the physio-somatic symptoms of somatization, depression, and chronic fatigue syndrome;Anderson;Acta Psychiatr. Scand.,2014

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