Inhibition of collapsin response mediator protein-2 phosphorylation ameliorates motor phenotype of ALS model mice expressing SOD1G93A-Reference-Cited by-同舟云学术

Inhibition of collapsin response mediator protein-2 phosphorylation ameliorates motor phenotype of ALS model mice expressing SOD1G93A

Published:2019-02 Issue: Volume:139 Page:63-68
ISSN:0168-0102
Container-title:Neuroscience Research
language:en
Short-container-title:Neuroscience Research

Author:

Numata-Uematsu Yurika,Wakatsuki Shuji,Nagano Seiichi,Shibata Megumi,Sakai Kazuhisa,Ichinohe Noritaka^ORCID,Mikoshiba Katsuhiko,Ohshima Toshio,Yamashita Naoya^ORCID,Goshima Yoshiro,Araki Toshiyuki

Funder

Intramural Research Grant for Neurological and Psychiatric Disorders of NCNP

Takeda Science Foundation

Creation of Innovation Centers for Advanced Interdisciplinary Research Areas Program in the Project for Developing Innovation System from the MEXT

Publisher

Elsevier BV

Subject

General Medicine,General Neuroscience

Reference27 articles.

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3. Aggregation and motor neuron toxicity of an ALS-linked SOD1 mutant independent from wild-type SOD1;Bruijn;Science,1998

4. Collapsin response mediator protein-2 regulates neurite formation by modulating tubulin GTPase activity;Chae;Cell. Signal.,2009

5. Collapsin response mediator protein-2 hyperphosphorylation is an early event in Alzheimer’s disease progression;Cole;J. Neurochem.,2007

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